Tuesday, 27 May 2008

10 best foods for eyes

American nutritionist Joy Bauer has come up with a list of top 10 foods for those suffering from AMD. The criteria: foods that are rich in the antioxidants beta carotene, vitamin C, vitamin E, lutein and zeaxanthin, zinc and omega-3 fats.

Here are her top 10 picks:

1. Carrots
2. Bell peppers
3. Broccoli
4. Brussels sprouts
5. Ostrich
6. Turkey
7. Sweet potatoes
8. Spinach
9. Wild salmon

10. Sardines



Click here to read her explanation of the choices and even suggestions on how to cook it!

Friday, 9 May 2008

MDS Sharing Session

Topic: Hi, I have macular degeneration
Date: May 17, 2008 (Sat)
Time: 2pm to 4pm
Venue: Seminar Rm, Alexandra Hospital
How do you tell others that you have macular degeneration? What are some of the problems you face when sharing about your vision problems? Is it better to say it or hide it?

Come join us for an intimate afternoon of sharing over coffee and tea. Hear how others tell their family, friends and colleagues about their struggle with macular degeneration. And share your own experience in communicating about MD.
Register with Anne 6238-7387 or Shing Huei pehshinghuei@gmail.com

Thursday, 1 May 2008

Gene therapy breakthrough

For the first time, researchers have used gene therapy to improve the vision in patients who are virtually blind. The breakthrough is offering hope to hundreds of thousands of patients with vision impairment, including macular degeneration sufferers.

Even though the experiments were conducted on those with a rare form of blindness called Leber's congenital amaurosis, researchers believe the approach can ultimately be used for a broad spectrum of disorders, including retinitis pigmentosa and MD.

The experiments were done in both the United States and Britain. In London, three patients were treated, with no ill effects. Even though they did not improve their visual acuity, one of them, 18-year-old Steven Howarth (below), had improved light sensitivity, especially at night.
Before the operation, Howarth was made to walk through a maze. He kept bumping into things and was disoriented. Six months after treatment, he walked through as fast as a normal person!

In Pennsylvania, the results were more impressive. The three patients were treated in one eye each. After two weeks, all reported improvement. One of them could read 3 1/2 lines better on an eye chart.

"All three subjects are asking if they can have their other eye injected," said Dr. Katherine A. High of Children's Hospital of Philadelphia. "That's a pretty good indicator of its effectiveness."

Another researcher hailed the results as a "landmark" and the most important therapeutic discovery in four decades in the field of retinal dystrophies.

Dogs given the same treatment have remained stable for at least eight years.

Gene therapy is when a defective gene is replaced with a good one. In the case of the Leber's, both groups of researchers used a gene called RPE65 that is defective in many Leber's patients.

For MD, it is caused by other defective genes. But the treatment principle would be the same. Researchers would have to design a specific delivery vehicle, or vector, for each disorder bearing the proper gene.